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    Cytotec (Misoprostol) is a drug that was originally developed as a medicinal product to protect the cavity of the stomach and duodenum from excessively rapid reabsorption of the mucous protective layer. Cytotec stimulates the production of mucus and suppresses the synthesis of pepsin, which is the basis of gastric juice. The active ingredient in Cytotec is prostaglandin E1. Cytotec is commonly prescribed for the treatment of gastrointestinal ulcers (both with bleeding and without). This drug can also be prescribed to terminate a pregnancy, but only if the timing allows for such a procedure and under the supervision of a doctor. Cytotec can also be prescribed for a previously incomplete medical abortion. For example, in instances when the egg did not entirely come out, or vaginal bleeding did not come. MISOPROSTOL ADMINISTRATION TO WOMEN WHO ARE PREGNANT CAN CAUSE ABORTION, PREMATURE BIRTH, OR BIRTH DEFECTS. UTERINE RUPTURE HAS BEEN REPORTED WHEN MISOPROSTOL WAS ADMINISTERED IN PREGNANT WOMEN TO INDUCE LABOR OR TO INDUCE ABORTION BEYOND THE EIGHTH WEEK OF PREGNANCY. PATIENTS MUST BE ADVISED OF THE ABORTIFACIENT PROPERTY AND WARNED NOT TO GIVE THE DRUG TO OTHERS. Misoprostol should not be used for reducing the risk of NSAID-induced ulcers in women of childbearing potential unless the patient is at high risk of complications from gastric ulcers associated with use of the NSAID, or is at high risk of developing gastric ulceration. In such patients, misoprostol may be prescribed if the patient: analog. Misoprostol contains approximately equal amounts of the two diastereomers presented below with their enantiomers indicated by (±): Misoprostol is a water-soluble, viscous liquid chemically described as (±) methyl 11α, 16-dihydroxy-16-methyl-9-oxoprost-13E-en-1-oate. Each misoprostol tablet for oral administration contains either 100 mcg or 200 mcg of misoprostol and has the following inactive ingredients: Hydrogenated caster oil, hypromellose, microcrystalline cellose, and sodium starch glycolate. Misoprostol is extensively absorbed, and undergoes rapid de-esterification to its free acid, which is responsible for its clinical activity and, unlike the parent compound, is detectable in plasma.

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